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Index by author

October 01, 2016; Volume 37,Issue 10

  1. Wang, C.-W.

    1. HEAD & NECK
      Evaluating Instantaneous Perfusion Responses of Parotid Glands to Gustatory Stimulation Using High-Temporal-Resolution Echo-Planar Diffusion-Weighted Imaging
      T.-W. Chiu, Y.-J. Liu, H.-C. Chang, Y.-H. Lee, J.-C. Lee, K. Hsu, C.-W. Wang, J.-M. Yang, H.-H. Hsu and C.-J. Juan
      American Journal of Neuroradiology October 2016, 37 (10) 1909-1915; DOI: https://doi.org/10.3174/ajnr.A4852

  2. Wang, P.-N.

    1. EXTRACRANIAL VASCULAR
      Open Access
      Intervention versus Aggressive Medical Therapy for Cognition in Severe Asymptomatic Carotid Stenosis
      C.-J. Lin, F.-C. Chang, K.-H. Chou, P.-C. Tu, Y.-H. Lee, C.-P. Lin, P.-N. Wang and I.-H. Lee
      American Journal of Neuroradiology October 2016, 37 (10) 1889-1897; DOI: https://doi.org/10.3174/ajnr.A4798

  3. Wang, Y.

    1. EDITOR'S CHOICEADULT BRAIN
      Open Access
      Magnetic Susceptibility from Quantitative Susceptibility Mapping Can Differentiate New Enhancing from Nonenhancing Multiple Sclerosis Lesions without Gadolinium Injection
      Y. Zhang, S.A. Gauthier, A. Gupta, L. Tu, J. Comunale, G.C.-Y. Chiang, W. Chen, C.A. Salustri, W. Zhu and Y. Wang
      American Journal of Neuroradiology October 2016, 37 (10) 1794-1799; DOI: https://doi.org/10.3174/ajnr.A4856

      In 54 patients, new T2-weighted lesions were evaluated for enhancement on conventional T1-weighted imaging with gadolinium, and their susceptibility values were measured on quantitative susceptibility mapping. Eighty-six of 133 new lesions that were gadolinium-enhancing had relative susceptibility values significantly lower than those of nonenhancing lesions. Using susceptibility values to discriminate enhancing from nonenhancing lesions showed a sensitivity of 88.4% and specificity of 91.5%, with a cutoff value of 11.2 parts per billion for QSM.

    2. ADULT BRAIN
      Open Access
      In Vivo 7T MR Quantitative Susceptibility Mapping Reveals Opposite Susceptibility Contrast between Cortical and White Matter Lesions in Multiple Sclerosis
      W. Bian, E. Tranvinh, T. Tourdias, M. Han, T. Liu, Y. Wang, B. Rutt and M.M. Zeineh
      American Journal of Neuroradiology October 2016, 37 (10) 1808-1815; DOI: https://doi.org/10.3174/ajnr.A4830

  4. Weprin, B.

    1. PEDIATRICS
      MR Imaging of the Cervical Spine in Nonaccidental Trauma: A Tertiary Institution Experience
      R. Jacob, M. Cox, K. Koral, C. Greenwell, Y. Xi, L. Vinson, K. Reeder, B. Weprin, R. Huang and T.N. Booth
      American Journal of Neuroradiology October 2016, 37 (10) 1944-1950; DOI: https://doi.org/10.3174/ajnr.A4817

  5. Wong, M.

    1. EDITOR'S CHOICEHEAD & NECK
      Imaging Appearance of SMARCB1 (INI1)-Deficient Sinonasal Carcinoma: A Newly Described Sinonasal Malignancy
      D.R. Shatzkes, L.E. Ginsberg, M. Wong, A.H. Aiken, B.F. Branstetter, M.A. Michel and N. Aygun
      American Journal of Neuroradiology October 2016, 37 (10) 1925-1929; DOI: https://doi.org/10.3174/ajnr.A4841

      SMARCB1 (INI1) is a tumor-suppressor gene that has been implicated in a growing number of malignancies involving multiple anatomic sites, including the kidneys, soft tissues, and the CNS (See OMIM *601607). The authors describe a case series of 17 patients collected from 6 different centers to give a comprehensive description of the appearance of these tumors on CT, MR, and PET/CT studies. SMARCB1 (INI1)-deficient sinonasal carcinoma should be included in the differential diagnosis of a central sinonasal mass demonstrating aggressive imaging features, particularly when there is associated calcification.

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