TABLE 2:

Clinical characteristics of mismatch cases

Age (y)/Sex	Duration	TIAs	vDWI	vPWI	Echocardiography	Carotid Duplex Sonography	Angiography	MRA	MRI	Therapy	Etiology
64/M	12 h	4	6.1	18.4	TTE: Dilatation L atrium	L carotid occlusion	Severe L ICA stenosis	Absent L ICA	Cortical acute infarcts R MCA territory	CEA	LAA
56/M	20 min	2	14.9	26.8	TTE: Normal	Unremarkable	Not performed	Unremarkable	Acute subcortical infarct L MCA territory	BP increased, MAP >100 mm Hg; IV heparin + oral aspirin	CE
61/M	20 min	2	0	245.7	TEE: Global systolic dysfunction, inferior-wall akinesis, decreased ejection fraction (40%), moderate aortic atherosclerosis	Occlusion L CCA, 60% stenosis R ICA	Not performed	L ICA occlusion, stenosis of L MCA branches	2 small, old R MCA infarcts, mild periventricular leukoaraiosis	BP increased, IV heparin + oral aspirin	LAA
67/F	30 min	2	1.5	21.1	TTE: Normal	Severe ICA stenosis, flow reversal on ophthalmic artery	R carotid stenosis	Irregular supraclinoid L ICA	Scattered small acute infarcts, watershed distribution in R MCA	CEA	LAA
42/F	20 min	1	0	372	Not performed	Not performed	L MCA stenosis with distal occlusion	L MCA occlusion	Unremarkable	EC-IC bypass	LAA
71/F	4 h	3	5.5	44	TTE: Normal	Not performed	L MCA (M1) stenosis	L MCA (M1) stenosis	L posterior corona radiata acute ischemia	Warfarin	LAA
63/M	20 s	Multiple	0	28.2	Not performed	60% stenosis R ICA	Severe L cavernous ICA stenosis, tandem stenosis bilateral ACAs	L cavernous ICA stenosis, bilateral ACA stenosis	Old confluent subcortical areas of leukomalacia in both frontal lobes, moderate leukoaraiosis	Balloon angioplasty	LAA

Note.—BP indicates blood pressure; IV intravenous; TEE, transesophageal; TTE, transthoracic.