Article Figures & Data
Figures
- Fig 1.
Images in a 47-year-old woman with SAH.
A, Sagittal T1-weighted SE image (TR/TE, 584/12) shows increased signal intensity throughout the basilar cisterns, fourth ventricle, and spinal subarachnoid space (black arrowheads). This area of relatively high signal intensity extends through the cerebral aqueduct into the posterior aspect of the third ventricle, with a dependent level just anterior to the massa intermedia (white arrow). The anterior inferior aspect of the interhemispheric fissure also shows slightly increased signal intensity, and the cortical sulci are not visualized in this area (black arrows). The hypointensity that corresponds to the aneurysm (arrowhead), and the A2 segments of the anterior cerebral arteries (ACAs), are seen on the background of SAH.
B, Axial FLAIR image (TR/TE/TI, 6212/100/1800) shows high signal intensity of the posterior aspect of the third ventricle and in the interhemispheric fissure surrounding the ACAs (arrows). A small, nonspecific area of high signal intensity is incidentally found in the right frontal lobe.
C, Axial T2-weighted SE image (2008/80) at the same level as in B. The signal intensity in the posterior aspect of the third ventricle and in the interhemispheric fissure (arrows) is higher that that of other CSF-containing spaces (arrowheads).
D, Axial T2-weighted SE image at the adjacent lower level. The signal intensity of the posterior aspect of the third ventricle, around the ACAs, and in the supravermian cistern (black arrows) is higher than that of the lateral ventricles and the anterior portion of the third ventricle (arrowheads). The larger flow-void area posterior to the ACAs (white arrow) was proved to correspond to the ACoA aneurysm on a subsequent intraarterial angiogram (not shown).
E, Axial T2*-weighted GRE image (800/35; flip angle, 25°) at approximately the same level as in D. The signal intensities within the third ventricle are reversed compared with those in D. The posterior aspect of the third ventricle is of lower signal intensity. This hypointensity is mild, presumably due to small amount of deoxyhemoglobin, which is partly counterbalanced by extremely high T2 signal intensity.
F, Axial proton density-weighted SE image (2008/20) at the same level as in D. The signal intensity within the posterior portion of the third ventricle, in the interhemispheric fissure, and in the supravermian cistern, is approximately the same as that in the gray matter.
G, Sagittal contrast-enhanced T1-weighted SE image (584/12) shows pooling of gadolinium-based contrast agent just anterior to corpus callosum (arrow). Homogeneously low signal intensity of the CSF is present in the lateral ventricle. The image quality is compromised by motion.
