Article Figures & Data
Figures
- FIG 1.
Axial DWI (A) and T2-weighted imaging (B) showing disseminated leukoencephalopathy with associated microhemorrhages (C) seen on the susceptibility-weighted imaging. D, Postcontrast T1-weighted imaging shows patchy enhancement of most WM lesions, with the larger lesion in the left cerebral hemisphere also showing central nonenhancement, suggestive of breakdown of the BBB and WM necrosis.
- FIG 2.
A and B, Axial FLAIR and coronal T2-weighted images at baseline MR imaging (day 25) showing multiple confluent areas of leukoencephalopathy. C and D, These lesions progressed to show development of cystic cavitations in the WM (arrows) as well as interval prominence of the lateral and third ventricles, suggesting brain atrophy on follow-up MR imaging on day 75.
- FIG 3.
Axial DWI, T2-weighted, FLAIR (A–C), and coronal T2-weighted images (D) at baseline MR imaging (day 10) showing multiple patchy areas of restricted diffusion/cytotoxic edema in the WM. E–H, Axial corresponding follow-up MR imaging (day 75) demonstrates development of cystic cavitations in the previous patchy cytotoxic WM lesions (arrows) as well as interval prominence of the lateral and third ventricles, suggesting brain atrophy. Follow-up MR imaging also showed marked progression of more confluent WM lesions surrounding the patchy cystic lesions.
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